Alois Haromy Department of Medicine, Cardiology Division, Pulmonary Hypertension Program University of Alberta Biography Publications Institution JoVE Articles Alois Haromy has not added a biography. If you are Alois Haromy and would like to personalize this page please email our Author Liaison for assistance. Publications Pyruvate Dehydrogenase Inhibition by the Inflammatory Cytokine TNFα Contributes to the Pathogenesis of Pulmonary Arterial Hypertension Journal of Molecular Medicine (Berlin, Germany). Aug, 2011 | Pubmed ID: 21809123 The Role of Nogo and the Mitochondria-endoplasmic Reticulum Unit in Pulmonary Hypertension Science Translational Medicine. Jun, 2011 | Pubmed ID: 21697531 Fatty Acid Oxidation and Malonyl-CoA Decarboxylase in the Vascular Remodeling of Pulmonary Hypertension Science Translational Medicine. Aug, 2010 | Pubmed ID: 20702857 Dehydroepiandrosterone Reverses Systemic Vascular Remodeling Through the Inhibition of the Akt/GSK3-{beta}/NFAT Axis Circulation. Sep, 2009 | Pubmed ID: 19752325 Airway Delivery of Mesenchymal Stem Cells Prevents Arrested Alveolar Growth in Neonatal Lung Injury in Rats American Journal of Respiratory and Critical Care Medicine. Dec, 2009 | Pubmed ID: 19713449 Statin Therapy, Alone or with Rapamycin, Does Not Reverse Monocrotaline Pulmonary Arterial Hypertension: the Rapamcyin-atorvastatin-simvastatin Study American Journal of Physiology. Lung Cellular and Molecular Physiology. Oct, 2007 | Pubmed ID: 17675370 Phosphodiesterase Type 5 is Highly Expressed in the Hypertrophied Human Right Ventricle, and Acute Inhibition of Phosphodiesterase Type 5 Improves Contractility Circulation. Jul, 2007 | Pubmed ID: 17606845 The Nuclear Factor of Activated T Cells in Pulmonary Arterial Hypertension Can Be Therapeutically Targeted Proceedings of the National Academy of Sciences of the United States of America. Jul, 2007 | Pubmed ID: 17596340 Oxygen Activates the Rho/Rho-kinase Pathway and Induces RhoB and ROCK-1 Expression in Human and Rabbit Ductus Arteriosus by Increasing Mitochondria-derived Reactive Oxygen Species: a Newly Recognized Mechanism for Sustaining Ductal Constriction Circulation. Apr, 2007 | Pubmed ID: 17353442 A Mitochondria-K+ Channel Axis is Suppressed in Cancer and Its Normalization Promotes Apoptosis and Inhibits Cancer Growth Cancer Cell. Jan, 2007 | Pubmed ID: 17222789 An Abnormal Mitochondrial-hypoxia Inducible Factor-1alpha-Kv Channel Pathway Disrupts Oxygen Sensing and Triggers Pulmonary Arterial Hypertension in Fawn Hooded Rats: Similarities to Human Pulmonary Arterial Hypertension Circulation. Jun, 2006 | Pubmed ID: 16735674 Vascular Endothelial Growth Factor Gene Therapy Increases Survival, Promotes Lung Angiogenesis, and Prevents Alveolar Damage in Hyperoxia-induced Lung Injury: Evidence That Angiogenesis Participates in Alveolarization Circulation. Oct, 2005 | Pubmed ID: 16230500 Gene Therapy Targeting Survivin Selectively Induces Pulmonary Vascular Apoptosis and Reverses Pulmonary Arterial Hypertension The Journal of Clinical Investigation. Jun, 2005 | Pubmed ID: 15931388 Dichloroacetate Prevents and Reverses Pulmonary Hypertension by Inducing Pulmonary Artery Smooth Muscle Cell Apoptosis Circulation Research. Oct, 2004 | Pubmed ID: 15375007 Oxygen-sensitive Kv Channel Gene Transfer Confers Oxygen Responsiveness to Preterm Rabbit and Remodeled Human Ductus Arteriosus: Implications for Infants with Patent Ductus Arteriosus Circulation. Sep, 2004 | Pubmed ID: 15353504 İnsan internal meme arter (İMA) Transplantasyon ve Stent: In-Stent Restenozis Geliştirme Eğitim Bir İnsan Modeli Xiaoqin Hua1,2, Tobias Deuse1,2, Evangelos D. Michelakis3, Alois Haromy3, Phil S. Tsao4, Lars Maegdefessel4, Reinhold G. Erben5, Claudia Bergow5, Boris B. Behnisch6, Hermann Reichenspurner1,2, Robert C. Robbins7, Sonja Schrepfer1,2,7 1University Heart Center Hamburg, TSI-Lab, Germany, 2Cardiovascular Research Center, University of Hamburg, 3Department of Medicine, Cardiology Division, Pulmonary Hypertension Program, University of Alberta, 4Department of Medicine, Stanford University School of Medicine, 5Department of Biomedical Sciences, Institute of Physiology, Pathophysiology, and Biophysics, University of Veterinary Medicine, Vienna, 6Translumina GmbH, Hechingen, 7Department of Cardiothoracic Surgery, Stanford University School of Medicine Medicine
İnsan internal meme arter (İMA) Transplantasyon ve Stent: In-Stent Restenozis Geliştirme Eğitim Bir İnsan Modeli Xiaoqin Hua1,2, Tobias Deuse1,2, Evangelos D. Michelakis3, Alois Haromy3, Phil S. Tsao4, Lars Maegdefessel4, Reinhold G. Erben5, Claudia Bergow5, Boris B. Behnisch6, Hermann Reichenspurner1,2, Robert C. Robbins7, Sonja Schrepfer1,2,7 1University Heart Center Hamburg, TSI-Lab, Germany, 2Cardiovascular Research Center, University of Hamburg, 3Department of Medicine, Cardiology Division, Pulmonary Hypertension Program, University of Alberta, 4Department of Medicine, Stanford University School of Medicine, 5Department of Biomedical Sciences, Institute of Physiology, Pathophysiology, and Biophysics, University of Veterinary Medicine, Vienna, 6Translumina GmbH, Hechingen, 7Department of Cardiothoracic Surgery, Stanford University School of Medicine Medicine
