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Journal / Biology / Bristfälligt Pms2, ERCC1, Ku86, CcOI i Field…
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Biology

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JoVE Journal Biology
Bristfälligt Pms2, ERCC1, Ku86, CcOI i Field defekter under Progression till tjocktarmscancer
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Bristfälligt Pms2, ERCC1, Ku86, CcOI i Field defekter under Progression till tjocktarmscancer

Article DOI: 10.3791/1931-v • 28:16 min • July 28th, 2010
July 28th, 2010 •
1, 1, 1, 1, 1, 1, 2,3, 1,4, 3, 5, 5, 5, 1
1Department of Cell Biology and Anatomy, College of Medicine, University of Arizona, Tucson, 2Southern Arizona Veterans Affairs Health Care System, Tucson, AZ, 3Department of Surgery, College of Medicine, University of Arizona, Tucson, 4Biomedical Diagnostics and Research, Tucson, AZ, 5Department of Medicine, College of Medicine, University of Arizona, Tucson

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Minskad / frånvarande uttryck för Pms2 och / eller ERCC1 i hela kryptor är en vanlig händelse inom 10 cm på varje sida av kolon adenocarcinom, sannolikt med stöd av ett fält fel med höga mutability och progression till cancer. Brist på Ku86 eller CcOI är mycket mindre vanligt i dessa fält defekter.

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Cellbiologi DNA-reparation apoptos synfältsdefekt tjocktarmscancer Pms2 ERCC1 cytokrom c oxidas subenheten jag Ku86 immunohistokemi Cancer resektion
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