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Studium der Hypothalamus Insulin-Signal an peripheren Glukoseintoleranz mit einem kontinuierlichen Medikament Infusion System in das Gehirn der Maus
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Studium der Hypothalamus Insulin-Signal an peripheren Glukoseintoleranz mit einem kontinuierlichen Medikament Infusion System in das Gehirn der Maus

Article DOI: 10.3791/56410-v • 08:32 min • January 4th, 2018
January 4th, 2018 •
1,2, 1,2,3
1The Ph.D. Program for Neural Regenerative Medicine, College of Medical Science and Technology, Taipei Medical University and National Health Research, 2Graduate Institute of Neural Regenerative Medicine, College of Medical Science and Technology, Taipei Medical University, 3TMU research center for Neurotrauma and Neuroregeneration, College of Medical Science and Technology, Taipei Medical University

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Dieses Protokoll untersucht die Rolle der Chemokin (C-C-Motiv) Liganden 5 (CCL5) im Hypothalamus durch die Bereitstellung von Antagonist MetCCL5, in das Gehirn der Maus mit einem Mikro-osmotischen Pumpe Gehirn Infusion System. Diese vorübergehende Hemmung der CCL5 Aktivität unterbrochen Hypothalamus Insulin Signalisierung, Glukoseintoleranz und periphere systemische Insulinsensitivität führen.

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Medizin Ausgabe 131 Gehirn-Drug-Delivery Mikro-osmotischen Pumpe Insulin Toleranz Glukosestoffwechsel Chemokin (C-C-Motiv) Liganden 5 (CCL5) Diabetes Mellitus Typ 2 (T2DM)
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