PB

Padmamalini Baskaran

School of Pharmacy, University of Wyoming

Affiliated withUniversity of Wyoming

Research Area

Biography

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JoVE Journal Publications

ArticleTotal : 1	Year
Publication title Cited by 10	2017

Other Publications

Article	Year
Desensitization of soluble guanylyl cyclase, the NO receptor, by S-nitrosylation. Proceedings of the National Academy of Sciences of the United States of America\| PubMed ID: 17636120	2007
PAS-mediated dimerization of soluble guanylyl cyclase revealed by signal transduction histidine kinase domain crystal structure. The Journal of biological chemistry\| PubMed ID: 18006497	2008
Structure of cinaciguat (BAY 58-2667) bound to Nostoc H-NOX domain reveals insights into heme-mimetic activation of the soluble guanylyl cyclase. The Journal of biological chemistry\| PubMed ID: 20463019	2010
Aspartate 102 in the heme domain of soluble guanylyl cyclase has a key role in NO activation. Biochemistry\| PubMed ID: 21491881	2011
Inhibition of nitric oxide-stimulated vasorelaxation by carbon monoxide-releasing molecules. Arteriosclerosis, thrombosis, and vascular biology\| PubMed ID: 21836072	2011
Identification of residues in the heme domain of soluble guanylyl cyclase that are important for basal and stimulated catalytic activity. PloS one\| PubMed ID: 22096512	2011
Soluble guanylyl cyclase is a target of angiotensin II-induced nitrosative stress in a hypertensive rat model. American journal of physiology. Heart and circulatory physiology\| PubMed ID: 22730391	2012
Protein disulfide-isomerase interacts with soluble guanylyl cyclase via a redox-based mechanism and modulates its activity. The Biochemical journal\| PubMed ID: 23477350	2013
Effects of enzymatically inactive recombinant botulinum neurotoxin type A at the mouse neuromuscular junctions. Toxicon : official journal of the International Society on Toxinology\| PubMed ID: 23810945	2013
Cyclophilin A is required for angiotensin II-induced p47phox translocation to caveolae in vascular smooth muscle cells. Arteriosclerosis, thrombosis, and vascular biology\| PubMed ID: 23846495	2013
Acute and chronic effects of botulinum neurotoxin a on the mammalian neuromuscular junction. Muscle & nerve\| PubMed ID: 24218344	2014
Acetylation of cyclophilin A is required for its secretion and vascular cell activation. Cardiovascular research\| PubMed ID: 24293519	2014
Cyclophilin A is an important mediator of platelet function by regulating integrin αIIbβ3 bidirectional signalling. Thrombosis and haemostasis\| PubMed ID: 24429998	2014
Capsaicin modulates acetylcholine release at the myoneural junction. European journal of pharmacology\| PubMed ID: 25446918	2014
The TRPM8 protein is a testosterone receptor: I. Biochemical evidence for direct TRPM8-testosterone interactions. The Journal of biological chemistry\| PubMed ID: 25480783	2015
The TRPM8 protein is a testosterone receptor: II. Functional evidence for an ionotropic effect of testosterone on TRPM8. The Journal of biological chemistry\| PubMed ID: 25480785	2015
Capsaicin induces browning of white adipose tissue and counters obesity by activating TRPV1 channel-dependent mechanisms. British journal of pharmacology\| PubMed ID: 27174467	2016
Preparation and Evaluation of PLGA-Coated Capsaicin Magnetic Nanoparticles. Pharmaceutical research\| PubMed ID: 28326459	2017
Perturbation to Cholesterol at the Neuromuscular Junction confers botulinum neurotoxin A sensitivity to neonatal mice. Toxicological sciences : an official journal of the Society of Toxicology\| PubMed ID: 28645175	2017

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