Roger A. Bannister

Department of Medicine-Cardiology Division, University of Colorado School of Medicine

Affiliated withUniversity of Colorado School of Medicine

Research Area

Biography

Roger A. Bannister has not added Biography.
If you are Roger A. Bannister and would like to personalize this page please email our Author Liaison for assistance.

JoVE Journal Publications

ArticleTotal : 1	Year
Publication title Cited by 26	2017

Other Publications

Article	Year
Reconstituted slow muscarinic inhibition of neuronal (Ca(v)1.2c) L-type Ca2+ channels. Biophysical journal\| PubMed ID: 12496094	2002
Mapping sites of potential proximity between the dihydropyridine receptor and RyR1 in muscle using a cyan fluorescent protein-yellow fluorescent protein tandem as a fluorescence resonance en... The Journal of biological chemistry\| PubMed ID: 15280389	2004
Neurokinin 1 receptors trigger overlapping stimulation and inhibition of CaV2.3 (R-type) calcium channels. Molecular pharmacology\| PubMed ID: 17050807	2007
Bridging the myoplasmic gap: recent developments in skeletal muscle excitation-contraction coupling. Journal of muscle research and cell motility\| PubMed ID: 17899404	2007
The alpha(1S) III-IV loop influences 1,4-dihydropyridine receptor gating but is not directly involved in excitation-contraction coupling interactions with the type 1 ryanodine receptor. The Journal of biological chemistry\| PubMed ID: 18556650	2008
The monomeric G proteins AGS1 and Rhes selectively influence Galphai-dependent signaling to modulate N-type (CaV2.2) calcium channels. American journal of physiology. Cell physiology\| PubMed ID: 18815223	2008
The skeletal L-type Ca(2+) current is a major contributor to excitation-coupled Ca(2+) entry. The Journal of general physiology\| PubMed ID: 19114636	2009
Reassessing the molecular mechanism of beta-adrenergic stimulation of cardiac L-type Ca2+ current. Channels (Austin, Tex.)\| PubMed ID: 19535897	2009
Effects of inserting fluorescent proteins into the alpha1S II-III loop: insights into excitation-contraction coupling. The Journal of general physiology\| PubMed ID: 19564426	2009
The cardiac alpha(1C) subunit can support excitation-triggered Ca2+ entry in dysgenic and dyspedic myotubes. Channels (Austin, Tex.)\| PubMed ID: 19625771	2009
The 63rd Symposium of the Society of General Physiologists: "muscles" instead of squid at Woods Hole. The Journal of general physiology\| PubMed ID: 19948997	2009
A malignant hyperthermia-inducing mutation in RYR1 (R163C): consequent alterations in the functional properties of DHPR channels. The Journal of general physiology\| PubMed ID: 20479108	2010
A malignant hyperthermia-inducing mutation in RYR1 (R163C): alterations in Ca2+ entry, release, and retrograde signaling to the DHPR. The Journal of general physiology\| PubMed ID: 20479110	2010
Looking for answers to EC coupling's persistent questions. The Journal of general physiology\| PubMed ID: 20584887	2010
Properties of Na+ currents conducted by a skeletal muscle L-type Ca2+ channel pore mutant (SkEIIIK). Channels (Austin, Tex.)\| PubMed ID: 21406961	2011
A shortcut to a skeletal muscle DHPR knock-in? The Journal of physiology\| PubMed ID: 21965631	2011
Malignant hyperthermia susceptibility arising from altered resting coupling between the skeletal muscle L-type Ca2+ channel and the type 1 ryanodine receptor. Proceedings of the National Academy of Sciences of the United States of America\| PubMed ID: 22547813	2012
Triclosan impairs excitation-contraction coupling and Ca2+ dynamics in striated muscle. Proceedings of the National Academy of Sciences of the United States of America\| PubMed ID: 22891308	2012
Ca(V)1.1: The atypical prototypical voltage-gated Ca²⁺ channel. Biochimica et biophysica acta\| PubMed ID: 22982493	2013
Impaired gating of an L-Type Ca(2+) channel carrying a mutation linked to malignant hyperthermia. Biophysical journal\| PubMed ID: 23663834	2013
Depressed pacemaker activity of sinoatrial node myocytes contributes to the age-dependent decline in maximum heart rate. Proceedings of the National Academy of Sciences of the United States of America\| PubMed ID: 24128759	2013
RGK protein-mediated impairment of slow depolarization- dependent Ca2+ entry into developing myotubes. Channels (Austin, Tex.)\| PubMed ID: 24476902	2014
Functional assessment of three Rem residues identified as critical for interactions with Ca(2+) channel β subunits. Pflugers Archiv : European journal of physiology\| PubMed ID: 25771954	2015
Apparent lack of physical or functional interaction between CaV1.1 and its distal C terminus. The Journal of general physiology\| PubMed ID: 25779869	2015
Rem uncouples excitation-contraction coupling in adult skeletal muscle fibers. The Journal of general physiology\| PubMed ID: 26078055	2015
Bridging the myoplasmic gap II: more recent advances in skeletal muscle excitation-contraction coupling. The Journal of experimental biology\| PubMed ID: 26792328	2016
Distinct Components of Retrograde Ca(V)1.1-RyR1 Coupling Revealed by a Lethal Mutation in RyR1. Biophysical journal\| PubMed ID: 26910427	2016
Defining the MO's of RGK proteins. Channels (Austin, Tex.)\| PubMed ID: 27249780	2016
Progressive impairment of CaV1.1 function in the skeletal muscle of mice expressing a mutant type 1 Cu/Zn superoxide dismutase (G93A) linked to amyotrophic lateral sclerosis. Skeletal muscle\| PubMed ID: 27340545	2016