Sakeneh Zraika

Division of Metabolism, VA Puget Sound Health Care System

Affiliated withVA Puget Sound Health Care System University of Washington

Research Area

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JoVE Journal Publications

ArticleTotal : 1	Year
Publication title Cited by 2	2020

Other Publications

Article	Year
Islet amyloid formation is an important determinant for inducing islet inflammation in high-fat-fed human IAPP transgenic mice. Diabetologia\| PubMed ID: 24965964	2014
Effects of sex and hormone replacement therapy use on the prevalence of isolated impaired fasting glucose and isolated impaired glucose tolerance in subjects with a family history of type 2 ... Diabetes\| PubMed ID: 17130501	2006
Superiority of the Modification of Diet in Renal Disease equation over the Cockcroft-Gault equation in screening for impaired kidney function in Japanese Americans. Diabetes research and clinical practice\| PubMed ID: 17141914	2007
Glucose- and time-dependence of islet amyloid formation in vitro. Biochemical and biophysical research communications\| PubMed ID: 17222388	2007
Identification of the amyloid-degrading enzyme neprilysin in mouse islets and potential role in islet amyloidogenesis. Diabetes\| PubMed ID: 17259373	2007
Relationship of liver enzymes to insulin sensitivity and intra-abdominal fat. Diabetes care\| PubMed ID: 17666458	2007
Inhibition of glycosaminoglycan synthesis and protein glycosylation with WAS-406 and azaserine result in reduced islet amyloid formation in vitro. American journal of physiology. Cell physiology\| PubMed ID: 17804609	2007
Body mass index is associated with increased creatinine clearance by a mechanism independent of body fat distribution. The Journal of clinical endocrinology and metabolism\| PubMed ID: 19584179	2009
β-cell loss and β-cell apoptosis in human type 2 diabetes are related to islet amyloid deposition. The American journal of pathology\| PubMed ID: 21641386	2011
Matrix metalloproteinase-9 reduces islet amyloid formation by degrading islet amyloid polypeptide. The Journal of biological chemistry\| PubMed ID: 23229548	2013
Rosiglitazone treatment does not decrease amyloid deposition in transplanted islets from transgenic mice expressing human islet amyloid polypeptide. Transplantation proceedings\| PubMed ID: 23267797	2013
Neprilysin deficiency protects against fat-induced insulin secretory dysfunction by maintaining calcium influx. Diabetes\| PubMed ID: 23328128	2013
One year of sitagliptin treatment protects against islet amyloid-associated β-cell loss and does not induce pancreatitis or pancreatic neoplasia in mice. American journal of physiology. Endocrinology and metabolism\| PubMed ID: 23736544	2013
Determination of Optimal Sample Size for Quantification of β-Cell Area, Amyloid Area and β-Cell Apoptosis in Isolated Islets. The journal of histochemistry and cytochemistry : official journal of the Histochemistry Society\| PubMed ID: 26216141	2015
Matrix Metalloproteinase-9 Protects Islets from Amyloid-induced Toxicity. The Journal of biological chemistry\| PubMed ID: 26483547	2015
The S20G substitution in hIAPP is more amyloidogenic and cytotoxic than wild-type hIAPP in mouse islets. Diabetologia\| PubMed ID: 27393137	2016
Improved glycaemia in high-fat-fed neprilysin-deficient mice is associated with reduced DPP-4 activity and increased active GLP-1 levels. Diabetologia\| PubMed ID: 27933334	2017
High fat feeding unmasks variable insulin responses in male C57BL/6 mouse substrains. The Journal of endocrinology\| PubMed ID: 28138002	2017
Neprilysin Is Required for Angiotensin-(1-7)'s Ability to Enhance Insulin Secretion via Its Proteolytic Activity to Generate Angiotensin-(1-2). Diabetes\| PubMed ID: 28559246	2017
Apoptosis Repressor With Caspase Recruitment Domain Ameliorates Amyloid-Induced β-Cell Apoptosis and JNK Pathway Activation. Diabetes\| PubMed ID: 28729244	2017
Neprilysin Deficiency Is Associated With Expansion of Islet β-Cell Mass in High Fat-Fed Mice. The journal of histochemistry and cytochemistry : official journal of the Histochemistry Society\| PubMed ID: 29553871	2018
Neprilysin inhibition: a new therapeutic option for type 2 diabetes? Diabetologia\| PubMed ID: 31089754	2019