Xiuhua Wang

Department of Surgery, University of Alberta

Affiliated withUniversity of Alberta

Research Area

Biography

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JoVE Journal Publications

ArticleTotal : 2	Year
Publication title	2022
Publication title Cited by 1	2022

Other Publications

Article	Year
Loss of TIMP3 enhances interstitial nephritis and fibrosis. Journal of the American Society of Nephrology : JASN\| PubMed ID: 19406980	2009
Type 1 diabetic cardiomyopathy in the Akita (Ins2WT/C96Y) mouse model is characterized by lipotoxicity and diastolic dysfunction with preserved systolic function. American journal of physiology. Heart and circulatory physiology\| PubMed ID: 19801494	2009
Loss of angiotensin-converting enzyme 2 accelerates maladaptive left ventricular remodeling in response to myocardial infarction. Circulation. Heart failure\| PubMed ID: 19808375	2009
TIMP2 deficiency accelerates adverse post-myocardial infarction remodeling because of enhanced MT1-MMP activity despite lack of MMP2 activation. Circulation research\| PubMed ID: 20056917	2010
Early activation of matrix metalloproteinases underlies the exacerbated systolic and diastolic dysfunction in mice lacking TIMP3 following myocardial infarction. American journal of physiology. Heart and circulatory physiology\| PubMed ID: 20675565	2010
Lack of tissue inhibitor of metalloproteinases 2 leads to exacerbated left ventricular dysfunction and adverse extracellular matrix remodeling in response to biomechanical stress. Circulation\| PubMed ID: 21986284	2011
Loss of Timp3 gene leads to abdominal aortic aneurysm formation in response to angiotensin II. The Journal of biological chemistry\| PubMed ID: 23144462	2012
TIMP3 is the primary TIMP to regulate agonist-induced vascular remodelling and hypertension. Cardiovascular research\| PubMed ID: 23524300	2013
TIMP2 and TIMP3 have divergent roles in early renal tubulointerstitial injury. Kidney international\| PubMed ID: 23760282	2014
Differential role of TIMP2 and TIMP3 in cardiac hypertrophy, fibrosis, and diastolic dysfunction. Cardiovascular research\| PubMed ID: 24692173	2014
Myocardial recovery from ischemia-reperfusion is compromised in the absence of tissue inhibitor of metalloproteinase 4. Circulation. Heart failure\| PubMed ID: 24842912	2014
Divergent roles of matrix metalloproteinase 2 in pathogenesis of thoracic aortic aneurysm. Arteriosclerosis, thrombosis, and vascular biology\| PubMed ID: 25657308	2015
Cardiomyocyte A Disintegrin And Metalloproteinase 17 (ADAM17) Is Essential in Post-Myocardial Infarction Repair by Regulating Angiogenesis. Circulation. Heart failure\| PubMed ID: 26136458	2015
Inhibition of Soluble Epoxide Hydrolase Limits Mitochondrial Damage and Preserves Function Following Ischemic Injury. Frontiers in pharmacology\| PubMed ID: 27375480	2016
A Disintegrin and Metalloprotease-17 Regulates Pressure Overload-Induced Myocardial Hypertrophy and Dysfunction Through Proteolytic Processing of Integrin β1. Hypertension (Dallas, Tex. : 1979)\| PubMed ID: 27550917	2016
Genetic deletion of soluble epoxide hydrolase provides cardioprotective responses following myocardial infarction in aged mice. Prostaglandins & other lipid mediators\| PubMed ID: 28104457	2017
Tissue Inhibitor of Matrix Metalloproteinase-1 Promotes Myocardial Fibrosis by Mediating CD63-Integrin β1 Interaction. Hypertension (Dallas, Tex. : 1979)\| PubMed ID: 28373589	2017
Myocardial overexpression of TIMP3 after myocardial infarction exerts beneficial effects by promoting angiogenesis and suppressing early proteolysis. American journal of physiology. Heart and circulatory physiology\| PubMed ID: 28550172	2017
Absence of Tissue Inhibitor of Metalloproteinase-4 (TIMP4) ameliorates high fat diet-induced obesity in mice due to defective lipid absorption. Scientific reports\| PubMed ID: 28740132	2017