Koh-ichi Nagata

Department of Molecular Neurobiology, Aichi Developmental Disability Center

Affiliated withAichi Developmental Disability Center

Research Area

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JoVE Journal Publications

ArticleTotal : 1	Year
Publication title Cited by 2	2024

Other Publications

Article	Year
Morphological characterization of mammalian timeless in the mouse brain development. Neuroscience research\| PubMed ID: 25448545	2015
Morphological characterization of Class III phosphoinositide 3-kinase during mouse brain development. Medical molecular morphology\| PubMed ID: 26242203	2016
Schizophrenia susceptibility gene product dysbindin-1 regulates the homeostasis of cyclin D1. Biochimica et biophysica acta\| PubMed ID: 27130439	2016
Role of Class III phosphoinositide 3-kinase in the brain development: possible involvement in specific learning disorders. Journal of neurochemistry\| PubMed ID: 27607605	2016
Autism spectrum disorder-associated genes and the development of dentate granule cells. Medical molecular morphology\| PubMed ID: 28534217	2017
Expression analyses of Phactr1 (phosphatase and actin regulator 1) during mouse brain development. Neuroscience research\| PubMed ID: 28803787	2018
MUNC18-1 gene abnormalities are involved in neurodevelopmental disorders through defective cortical architecture during brain development. Acta neuropathologica communications\| PubMed ID: 29191246	2017
Expression analyses of Dusp22 (Dual-specificity phosphatase 22) in mouse tissues. Medical molecular morphology\| PubMed ID: 29282540	2018
Functions of Rhotekin, an Effector of Rho GTPase, and Its Binding Partners in Mammals. International journal of molecular sciences\| PubMed ID: 30037057	2018
Role of Per3, a circadian clock gene, in embryonic development of mouse cerebral cortex. Scientific reports\| PubMed ID: 30971765	2019
Expression Analyses of POGZ, A Responsible Gene for Neurodevelopmental Disorders, during Mouse Brain Development. Developmental neuroscience\| PubMed ID: 31430754	2019
Spatiotemporally Dependent Vascularization Is Differently Utilized among Neural Progenitor Subtypes during Neocortical Development. Cell reports\| PubMed ID: 31665628	2019
Biochemical and morphological characterization of SEPT1 in mouse brain. Medical molecular morphology\| PubMed ID: 32146512	2020
Neuropathophysiological significance of the c.1449T>C/p.(Tyr64Cys) mutation in the CDC42 gene responsible for Takenouchi-Kosaki syndrome. Biochemical and biophysical research communications\| PubMed ID: 32819561	2020
Heterotrimeric G-protein, Gi1, is involved in the regulation of proliferation, neuronal migration, and dendrite morphology during cortical development in vivo. Journal of neurochemistry\| PubMed ID: 33025585	2021
Pogz deficiency leads to transcription dysregulation and impaired cerebellar activity underlying autism-like behavior in mice. Nature communications\| PubMed ID: 33203851	2020
Expression analyses of PLEKHG2, a Rho family-specific guanine nucleotide exchange factor, during mouse brain development. Medical molecular morphology\| PubMed ID: 33492483	2021
Expression Analyses of Mediator Complex Subunit 13-Like: A Responsible Gene for Neurodevelopmental Disorders during Mouse Brain Development. Developmental neuroscience\| PubMed ID: 33794529	2021
Author Correction: Pogz deficiency leads to transcription dysregulation and impaired cerebellar activity underlying autism-like behavior in mice. Nature communications\| PubMed ID: 34140510	2021
Physiological significance of WDR45, a responsible gene for β-propeller protein associated neurodegeneration (BPAN), in brain development. Scientific reports\| PubMed ID: 34799629	2021
The synaptic scaffolding protein CNKSR2 interacts with CYTH2 to mediate hippocampal granule cell development. The Journal of biological chemistry\| PubMed ID: 34800437	2021
Expression Analyses of Rac3, a Rho Family Small GTPase, during Mouse Brain Development. Developmental neuroscience\| PubMed ID: 34839287	2022
Pathophysiological Mechanisms in Neurodevelopmental Disorders Caused by Rac GTPases Dysregulation: What's behind Neuro-RACopathies. Cells\| PubMed ID: 34943902	2021
Functions of CNKSR2 and Its Association with Neurodevelopmental Disorders. Cells\| PubMed ID: 35053419	2022
Impaired Function of PLEKHG2, a Rho-Guanine Nucleotide-Exchange Factor, Disrupts Corticogenesis in Neurodevelopmental Phenotypes. Cells\| PubMed ID: 35203342	2022
Expression Analyses of Cep152, a Responsible Gene Product for Autosomal Recessive Primary Microcephaly, during Mouse Brain Development. Developmental neuroscience\| PubMed ID: 35259752	2022
Gain-of-function p.F28S variant in disrupts neuronal differentiation, migration and axonogenesis during cortical development, leading to neurodevelopmental disorder. Journal of medical genetics\| PubMed ID: 35595279	2023
Variant-specific changes in RAC3 function disrupt corticogenesis in neurodevelopmental phenotypes. Brain : a journal of neurology\| PubMed ID: 35851598	2022
Expression Analyses of Polo-Like Kinase 4, a Gene Product Responsible for Autosomal Recessive Microcephaly and Seckel Syndrome, during Mouse Brain Development. Developmental neuroscience\| PubMed ID: 36067731	2022
Deficiency of , a gene related to intellectual disability, causes impaired neuronal development and a mild behavioural phenotype. Brain communications\| PubMed ID: 36106092	2022
Erratic and blood vessel-guided migration of astrocyte progenitors in the cerebral cortex. Nature communications\| PubMed ID: 36323680	2022
Pathophysiological Mechanism of Neurodevelopmental Disorders-Overview. Cells\| PubMed ID: 36552846	2022
Expression Analyses of Rich2/Arhgap44, a Rho Family GTPase-Activating Protein, during Mouse Brain Development. Developmental neuroscience\| PubMed ID: 36630934	2023
MED13L and its disease-associated variants influence the dendritic development of cerebral cortical neurons in the mammalian brain. Journal of neurochemistry\| PubMed ID: 36798993	2023
Histological Analysis of a Mouse Model of the 22q11.2 Microdeletion Syndrome. Biomolecules\| PubMed ID: 37238632	2023
A missense variant at the RAC1-PAK1 binding site of RAC1 inactivates downstream signaling in VACTERL association. Scientific reports\| PubMed ID: 37328543	2023
Expression analyses of WAC, a responsible gene for neurodevelopmental disorders, during mouse brain development. Medical molecular morphology\| PubMed ID: 37402055	2023
Expression analysis of type I ARF small GTPases ARF1-3 during mouse brain development. Molecular biology reports\| PubMed ID: 38227057	2024