Florent Elefteriou

Department of Pharmacology, Vanderbilt University

Research Area

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JoVE Journal Publications

ArticleTotal : 1	Year
Publication title Cited by 115	2012

Other Publications

Article	Year
Leptin regulates bone formation via the sympathetic nervous system. Cell\| PubMed ID: 12419242	2002
Common endocrine control of body weight, reproduction, and bone mass. Annual review of nutrition\| PubMed ID: 12730321	2003
Reversible skeletal abnormalities in gamma-glutamyl transpeptidase-deficient mice. Endocrinology\| PubMed ID: 12810527	2003
[Control of body mass by leptin occurs through the sympathetic nervous system]. MÃ©decine sciences : M/S\| PubMed ID: 12836204	2003
Monosodium glutamate-sensitive hypothalamic neurons contribute to the control of bone mass. Endocrinology\| PubMed ID: 12933656	2003
Essential role of STAT3 in body weight and glucose homeostasis. Molecular and cellular biology\| PubMed ID: 14673160	2004
Enhanced bone formation in lipodystrophic PPARgamma(hyp/hyp) mice relocates haematopoiesis to the spleen. EMBO reports\| PubMed ID: 15359271	2004
Leptin regulation of bone resorption by the sympathetic nervous system and CART. Nature\| PubMed ID: 15724149	2005
Boning up on ephrin signaling. Cell\| PubMed ID: 16901775	2006
ATF4 mediation of NF1 functions in osteoblast reveals a nutritional basis for congenital skeletal dysplasiae. Cell metabolism\| PubMed ID: 17141628	2006
Regulation of bone remodeling by the central and peripheral nervous system. Archives of biochemistry and biophysics\| PubMed ID: 18410742	2008
Cocaine and amphetamine-regulated transcript may regulate bone remodeling as a circulating molecule. Endocrinology\| PubMed ID: 18450969	2008
Vimentin inhibits ATF4-mediated osteocalcin transcription and osteoblast differentiation. The Journal of biological chemistry\| PubMed ID: 19726676	2009
Skeletal abnormalities in neurofibromatosis type 1: approaches to therapeutic options. American journal of medical genetics. Part A\| PubMed ID: 19764036	2009
Atf4 regulates chondrocyte proliferation and differentiation during endochondral ossification by activating Ihh transcription. Development (Cambridge, England)\| PubMed ID: 19906842	2009
Local low-dose lovastatin delivery improves the bone-healing defect caused by Nf1 loss of function in osteoblasts. Journal of bone and mineral research : the official journal of the American Society for Bone and Mineral Research\| PubMed ID: 20200958	2010
Î²2-Adrenergic receptor signaling in osteoblasts contributes to the catabolic effect of glucocorticoids on bone. Endocrinology\| PubMed ID: 21266510	2011
Mice lacking Nf1 in osteochondroprogenitor cells display skeletal dysplasia similar to patients with neurofibromatosis type I. Human molecular genetics\| PubMed ID: 21757497	2011
Genetic mouse models for bone studies--strengths and limitations. Bone\| PubMed ID: 21907838	2011
Chondrocytic Atf4 regulates osteoblast differentiation and function via Ihh. Development (Cambridge, England)\| PubMed ID: 22190639	2012
Stimulation of host bone marrow stromal cells by sympathetic nerves promotes breast cancer bone metastasis in mice. PLoS biology\| PubMed ID: 22815651	2012