Homonymous Hemianopia
Homonymous Hemianopia - Related Videos
11:12
Driving Simulation in the Clinic: Testing Visual Exploratory Behavior in Daily Life Activities in Patients with Visual Field Defects
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Cited by 20 •
2012
Patients with visual deficits after stroke report about different constraints in daily life most likely due to variable compensatory strategies, which are difficult to differentiate in clinical routine. We present a clinical set-up which allows measurement of different compensatory head- and eye-movement-strategies and evaluating their effects on driving performance.
06:19
Eye Tracking in Cognitive Experiments
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2023
Eye tracking as the name suggests involves tracking of eye-movements. It is a non-invasive, sensitive tool that quantifies and measures eye-movements to describe an individuals' cognitive state. An eye-movement between two fixation points is called a saccade, which is one of the fastest motor movements in our body. By observing the profiles of these eye movements, scientists can better understand neural deficits in patients with cognitive impairments. In this video, we will first look at an...
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Lesion localization links the visual deficit to a particular segment of the retrochiasmal pathway. Damage may involve the optic tract, optic radiations, or primary visual cortex, and the resulting field pattern helps clinicians determine where transmission has been interrupted. Neuroimaging then provides anatomical evidence, supporting evaluation of stroke, tumor, traumatic injury, or another brain disorder.
Because the affected pathway carries information from the opposite side of space, disruption on one side of the brain can impair awareness of that corresponding side in both eyes. This organization explains why the deficit is neurologic rather than an isolated eye problem. It also guides clinicians toward brain-based assessment and imaging rather than relying only on ocular examination.
Stroke is a common cause, but the same pathway can be disrupted by tumors, traumatic injury, or other brain disorders. These causes differ in their underlying disease process, yet they can produce a comparable visual-field problem because each may interrupt signal transmission within the retrochiasmal pathway. Identifying the cause is therefore essential to interpreting the deficit in its neurological context.
Reading, navigation, mobility, and other daily activities become difficult because visual information from part of surrounding space is no longer reliably available to guide attention and movement. Scanning training addresses this limitation by encouraging deliberate search of the affected side, while compensatory strategies and optical aids can support safer task performance and greater functional independence.
Visual field testing identifies the affected portion of visual space and helps characterize the deficit for clinical evaluation. Combined with neuroimaging, it can localize interruption within the optic tract, optic radiations, or primary visual cortex. That localization helps relate test findings to functional problems and guide compensatory support.
Neuroimaging complements visual field testing by showing whether a structural brain abnormality could account for the deficit. In this context, imaging helps investigate recognized causes such as stroke, tumor, or traumatic injury and may support localization within the retrochiasmal pathway. Used together, the tests connect anatomical findings with the measured visual-field pattern.
Rehabilitation focuses on practical compensation rather than simply describing the missing field. Scanning training teaches a deliberate visual search strategy, and broader compensatory strategies can be incorporated into reading, navigation, mobility, and daily tasks. Optical aids provide another option. These approaches are intended to improve safety and functional independence when the deficit interferes with ordinary activities.