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En rotte model af trykoverbelastning induceret moderat remodellering og systolisk dysfunktion i modsætning til Åbenlys systolisk hjertesvigt
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En rotte model af trykoverbelastning induceret moderat remodellering og systolisk dysfunktion i modsætning til Åbenlys systolisk hjertesvigt

Article DOI: 10.3791/60954-v • 07:13 min • April 30th, 2020
April 30th, 2020 •
1, 2, 1,2,3
1Department of Medicine/Heart and Vascular Institute, Tulane University, 2Department of Physiology, Tulane University, 3Department of Pharmacology, Tulane University

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Vi beskriver oprettelsen af en rotte model af trykoverbelastning induceret moderat remodellering og tidlig systolisk dysfunktion, hvor signal transduktionveje involveret i indledningen af remodeling processen er aktiveret. Dette dyr model vil støtte i at identificere molekylære mål for anvendelse af tidlig terapeutisk anti-remodeling strategier for hjertesvigt.

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Medicin Rotte Trykoverbelastning Hypertrofi Hjertesvigt Remodellering Signal Transduktion Energi Metabolisme Calcium Cycling
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