Stephen J. Crocker

Stephen J. Crocker

Department of Neuroscience, University of Connecticut Health Center

Affiliated withUniversity of Connecticut Health Center

Research Area

Biography

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JoVE Journal Publications

ArticleTotal : 1
Year
An <em>In Vitro</em> Model for the Study of Cellular Pathophysiology in Globoid Cell Leukodystrophy
Publication title

Cited by 3

2014

Other Publications

Article
Year
Inhibition of calpains prevents neuronal and behavioral deficits in an MPTP mouse model of Parkinson's disease.

The Journal of neuroscience : the official journal of the Society for Neuroscience| PubMed ID: 12764095

2003
Cyclin-dependent kinase 5 is a mediator of dopaminergic neuron loss in a mouse model of Parkinson's disease.

Proceedings of the National Academy of Sciences of the United States of America| PubMed ID: 14595022

2003
The TIMPs tango with MMPs and more in the central nervous system.

Journal of neuroscience research| PubMed ID: 14689443

2004
Regulation of dopaminergic loss by Fas in a 1-methyl-4-phenyl-1,2,3,6-tetrahydropyridine model of Parkinson's disease.

The Journal of neuroscience : the official journal of the Society for Neuroscience| PubMed ID: 14985447

2004
2004
2006
2006
2006
2007
2007
2007
2008
2009
2009
How factors secreted from astrocytes impact myelin repair.

Journal of neuroscience research| PubMed ID: 20857501

2011
2011
Astrocytic tissue inhibitor of metalloproteinase-1 (TIMP-1) promotes oligodendrocyte differentiation and enhances CNS myelination.

The Journal of neuroscience : the official journal of the Society for Neuroscience| PubMed ID: 21508247

2011
2011
An alternate perspective on the roles of TIMPs and MMPs in pathology.

The American journal of pathology| PubMed ID: 22033229

2012
2012
2013
2014
2013
2014
2014