Tachycardia
Tachycardia - Related Videos
10:08
Tachycardia-Induced Cardiomyopathy As a Chronic Heart Failure Model in Swine
0 Views •
Cited by 13 •
2018
Here, we present a protocol to produce tachycardia-induced cardiomyopathy in swine. This model represents a potent way to study the hemodynamics of progressive chronic heart failure and the effects of applied treatment.
06:57
Ablation of Ischemic Ventricular Tachycardia Using a Multipolar Catheter and 3-dimensional Mapping System for High-density Electro-anatomical Reconstruction
0 Views •
2019
With the following protocol, we provide an approach to Ventricular Tachycardia (VT) ablation using high density mapping with a multipolar catheter and 3D mapping system enhancing the success of the procedure.
09:36
Dual-Dye Optical Mapping of Hearts from RyR2R2474S Knock-In Mice of Catecholaminergic Polymorphic Ventricular Tachycardia
0 Views •
Cited by 1 •
2023
This protocol introduces dual-dye optical mapping of mouse hearts obtained from wild-type and knock-in animals affected by catecholaminergic polymorphic ventricular tachycardia, including electrophysiological measurements of transmembrane voltage and intracellular Ca2+ transients with high temporal and spatial resolution.
08:33
Porcine Cardiac Arrest Model Using an Implantable Defibrillator
0 Views •
2026
This protocol demonstrates the use of an implantable cardioverter-defibrillator (ICD) to reliably induce ventricular arrhythmias, perform internal cardioversion, and obtain intracardiac ECG recordings in a porcine model.
12:09
Patient-specific Modeling of the Heart: Estimation of Ventricular Fiber Orientations
0 Views •
Cited by 14 •
2013
A methodology to estimate ventricular fiber orientations from in vivo images of patient heart geometries for personalized modeling is described. Validation of the methodology performed using normal and failing canine hearts demonstrate that that there are no significant differences between estimated and acquired fiber orientations at a clinically observable level.
FAQs
Different electrical mechanisms produce different tachycardia patterns and clinical implications. Reentry results from an electrical impulse circulating through a pathway, whereas enhanced automaticity reflects increased spontaneous impulse generation. Triggered activity arises from abnormal impulses linked to preceding electrical events. Distinguishing these mechanisms helps clinicians interpret an electrocardiogram and connect the rhythm to an appropriate management strategy.
The heart may accelerate because the sinus node responds appropriately to exercise, fever, or stress, or because abnormal electrical activity disrupts the normal rhythm. This distinction matters because treating a temporary physiological response differs from evaluating a rhythm caused by reentry, enhanced automaticity, or triggered activity. Clinical findings and electrocardiographic assessment help separate these possibilities.
Tachycardia may reflect an underlying problem rather than an isolated electrical abnormality. Anemia, infection, medication effects, and heart disease can contribute to a faster rate or alter cardiac rhythm. Identifying these contributors is important because management may focus on correcting the precipitating condition, controlling the rate, restoring normal rhythm, or addressing the cardiac disorder.
Evaluation combines symptoms, vital signs, and an electrocardiogram with a search for possible causes. Symptoms and vital signs help indicate how the rhythm is affecting the patient, while the electrocardiogram helps identify the rhythm itself. Clinicians also consider infection, anemia, medication effects, and heart disease so that the observed rate is interpreted in context.
An electrocardiogram helps clinicians recognize whether rapid activity reflects sinus node acceleration or an abnormal rhythm involving the atria or ventricles. That distinction supports decisions about whether to treat an underlying trigger, control the rate, or attempt to restore normal rhythm. It also provides essential context when symptoms or vital signs suggest that circulation may be compromised.
Urgency depends less on the rate alone than on its effect on circulation and the patient’s overall clinical condition. Evaluation becomes urgent when circulation is compromised, because clinicians may need to act promptly while identifying the rhythm and cause. Management can include treating the trigger, controlling the rate, restoring normal rhythm, or providing immediate supportive care.