Neoplastic Polyps
Neoplastic Polyps - Related Videos
09:58
Investigating the Three-dimensional Flow Separation Induced by a Model Vocal Fold Polyp
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Cited by 2 •
2014
Vocal fold polyps can disrupt vocal fold dynamics and thus can have devastating consequences on a patient's ability to communicate. Three-dimensional flow separation induced by a wall-mounted model polyp and its impact on the wall pressure loading are examined using particle image velocimetry, skin friction line visualization, and wall pressure measurements.
09:05
Generation and Quantitative Analysis of Pulsed Low Frequency Ultrasound to Determine the Sonic Sensitivity of Untreated and Treated Neoplastic Cells
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2015
Selective damage of human leukemia cells can be achieved through a novel approach of applying low frequency ultrasound both with and without chemotherapeutic pretreatment of leukemic and normal hematopoietic cells.
05:15
Grossing of Non-neoplastic Globes, Including Fetal Eyes
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Cited by 1 •
2025
Thorough grossing and standardized sectioning of non-neoplastic globes are important for obtaining high-quality reproducible histopathologic sections. Guidelines for a standardized grossing of adult and fetal non-neoplastic globes are provided herein. Different options for globe sectioning are demonstrated and discussed, including criteria for case-based decision-making.
03:01
Improved Hysteroscopic Resection of Endometrial Polyps Using 6-Fr Micro-Scissors and Forceps
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2024
The novel technique presented here employs a combination of 6-Fr micro-scissors and forceps for hysteroscopic treatment of endometrial polyps, demonstrating encouraging outcomes for infertile patients afflicted with this condition.
07:23
Inducing Polyp Bail-out in Coral Colonies to Obtain Individualized Micropropagates for Laboratory Experimental Use
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Cited by 3 •
2022
Polyp bail-out is a process induced by acute stress, in which coral polyps digest the tissue connecting them to their colony and detach from it to live as individuals. The present protocol describes how to induce coral micropropagation by bail-out using hypersaline or calcium-free seawater treatments.
FAQs
Acquired genetic and epigenetic changes can disrupt the normal coordination of epithelial cell division, differentiation, and tissue architecture. Rather than producing a transient reactive change, these alterations support the persistence and expansion of abnormal cells within a localized growth. This mechanism explains why examining cellular features is important when assessing the lesion’s biological significance.
Dysplasia reflects abnormal cellular and architectural features within an adenoma and helps characterize how far the tissue has departed from normal organization. Because some adenomas can participate in the adenoma–carcinoma sequence, documenting the degree of dysplasia provides clinically relevant information. Histopathology therefore adds biological interpretation to the lesion’s appearance during colonoscopy.
Their significance lies in the combination of a localized epithelial growth with acquired changes that affect cell division, differentiation, and tissue organization. This distinguishes the process from an isolated change in shape or architecture without a clonal cellular basis. In medicine, that distinction matters because selected lesions may progress toward colorectal cancer and warrant structured evaluation.
Colonoscopy identifies the lesion and documents features including its size, number, and morphology. These observations describe the distribution and visible structure of the growths before or during endoscopic management. Recording them creates a clinical basis for subsequent histopathologic correlation and helps guide decisions about removal and follow-up surveillance.
Histopathologic examination evaluates the lesion’s cellular features and architectural changes, including findings relevant to dysplasia. This microscopic assessment complements colonoscopy, which primarily shows visible characteristics such as size and morphology. Together, the two approaches provide a more complete characterization of the lesion and support decisions about endoscopic removal and subsequent surveillance.
Their clinical importance comes from the opportunity to interrupt the adenoma–carcinoma sequence through identification, endoscopic removal, and appropriate surveillance. This approach can prevent a recognized precursor lesion from continuing along a cancer-associated pathway. Scientifically, these polyps also provide a model for studying early tumor development, including the genetic, epigenetic, and architectural changes that arise before cancer.