Anastomotic Stricture
Anastomotic Stricture - Related Videos
04:05
Endoscopic Vacuum Therapy for the Treatment of Anastomotic Leakage after Total Gastrectomy with Esophagojejunostomy
0 Views •
2025
Here, we present a protocol for endoscopic vacuum therapy (EVT) in the management of anastomotic leaks following total gastrectomy with esophagojejunostomy.
05:57
ADSC-sheet Transplantation to Prevent Stricture after Extended Esophageal Endoscopic Submucosal Dissection
0 Views •
Cited by 14 •
2017
This study reports a successful method of endoscopical adipose tissue-derived stromal cell (ADSC)-sheet transplantation for esophageal stricture prevention after an extended endoscopic submucosal dissection (ESD) in a swine model.
01:30
Esophageal Strictures-I: Introduction
0 Views •
2025
Esophageal strictures involve abnormal narrowing or tightening of the esophagus. They vary in length and severity, ranging from mild constriction to complete obstruction, and are classified as benign (noncancerous) or malignant (cancerous). Etiology The primary cause of esophageal strictures is long-standing gastroesophageal reflux disease (GERD), accounting for about 70 to 80% of adult cases. Chronic acid reflux can lead to injury and scarring of the esophageal lining, culminating in...
05:09
Urethral Stricture Induction Followed by Buccal Mucosa Graft Urethroplasty in a Rat Model
0 Views •
Cited by 3 •
2023
In the present protocol, a urethral stricture induction was developed in Wistar rats, followed by urethral reconstruction with a buccal mucosa graft. A retrograde urethrogram and laser Doppler assessment were performed, validating urethral reconstruction (after stricture formation) and graft placement.
05:42
Development of a Murine Model for Femoral Artery Anastomotic Stenosis
0 Views •
2025
Here we present a murine model for femoral artery anastomosis, offering researchers a valuable animal model to study and simulate vascular anastomotic stenosis. This development is crucial for advancing our understanding of the pathophysiology underlying this condition and facilitating more accurate and effective research into vascular diseases.
FAQs
Excessive scar formation during healing can progressively reduce lumen diameter at the surgical connection. As the passage becomes narrower, food, fluids, or intestinal contents may move less effectively. Ischemia, inflammation, infection, and technical factors can intensify this process or contribute independently, helping explain why strictures vary in severity and clinical presentation.
Ischemia, inflammation, infection, and technical factors can alter healing at the connection rather than simply reflecting the amount of scar already present. The overview identifies scar formation as the usual cause, while these conditions provide additional mechanisms for lumen loss. Recognizing this combination matters because more than one factor may contribute to the narrowing.
Symptoms depend partly on where the narrowing develops and which contents normally pass through that connection. Affected patients may experience dysphagia, abdominal pain, vomiting, bloating, or altered bowel function. This variation allows the clinical presentation to reflect the involved portion of the digestive system rather than producing one uniform symptom pattern.
Clinicians may confirm the narrowing with endoscopy or imaging. These approaches provide evidence that the lumen at the surgically created connection has become reduced and help relate the suspected obstruction to the patient’s symptoms. Confirmation is important before selecting management, particularly when symptoms could involve impaired passage of food, fluids, or intestinal contents.
Endoscopic balloon dilation is a commonly used treatment, but the overview notes that it may need to be repeated. This indicates that one intervention may not adequately address the narrowed passage in every case. Repeated dilation therefore remains part of management when the stricture continues to require treatment after an earlier endoscopic intervention.
Temporary stent placement may be combined with balloon dilation as an additional treatment option for the narrowed connection. When an anastomotic stricture is severe or recurrent, revision surgery may be required. These choices reflect escalation of management according to the persistence or seriousness of the narrowing and the response to endoscopic treatment.