Post-stroke Cognitive Impairment
Post-stroke Cognitive Impairment - Related Videos
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Brain Imaging Investigation of the Impairing Effect of Emotion on Cognition
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Cited by 11 •
2012
We present a protocol that allows investigation of the neural mechanisms mediating the detrimental impact of emotion on cognition, using functional magnetic resonance imaging. This protocol can be used with both healthy and clinical participants.
02:59
Stereotactic Injection of Hydrogel into a Post-Stroke Mouse Model
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2025
Source: Wilson, K.L., et al. Injection of Hydrogel Biomaterial Scaffolds to The Brain After Stroke. J. Vis. Exp. (2020)This video demonstrates the stereotaxic injection of a hydrogel into the stroke core of a post-stroke mouse, highlighting its role in scaffold formation that mimics the extracellular matrix, promotes cellular infiltration, and supports angiogenesis, underscoring its neurotherapeutic potential in stroke recovery.
07:35
Cognitive Function and Upper Limb Rehabilitation Training Post-Stroke Using a Digital Occupational Training System
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Cited by 7 •
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The current protocol outlines how the VR-based digital occupational training system enhances the rehabilitation of patients with cognitive impairment and upper limb dysfunction following a stroke.
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07:27
Autoradiographic Measurements of [14C]-Iodoantipyrine in Rat Brain Following Central Post-Stroke Pain
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Cited by 1 •
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We present a protocol to measure [14C]-iodoantipyrine (IAP) uptake and assess the activation of neural substrates that are involved in central post-stroke pain (CPSP) in a rodent model.
01:38
Cognitive Dissonance
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2020
Social psychologists have documented that feeling good about ourselves and maintaining positive self-esteem is a powerful motivator of human behavior (Tavris & Aronson, 2008). In the United States, members of the predominant culture typically think very highly of themselves and view themselves as good people who are above average on many desirable traits (Ehrlinger, Gilovich, & Ross, 2005). Often, our behavior, attitudes, and beliefs are affected when we experience a threat to our...
FAQs
Cerebrovascular injury can disrupt interconnected brain networks that support attention, memory, language, executive control, and visuospatial processing. Consequently, difficulties may appear across several cognitive domains or may be concentrated in one area, depending on which networks the stroke affects. This network-based perspective helps clinicians interpret patterns of impairment and plan rehabilitation around the person’s specific cognitive profile.
Lesion location helps determine which cognitive functions are vulnerable, while stroke severity indicates the broader impact of the cerebrovascular injury. Vascular risk factors and coexisting neurological or mood changes can further shape performance and recovery needs. Considering these influences prevents clinicians from assuming that all patients will show the same pattern or require identical assessment and support.
Poor performance on a cognitive task may reflect more than impaired thinking. Language difficulty can interfere with understanding or expressing responses, motor limitations can affect task completion, and neurological or mood changes can alter engagement and performance. Clinicians therefore combine the patient’s history with cognitive screening and, when needed, neuropsychological testing to characterize affected domains more accurately.
Evaluation begins with the patient’s history and a cognitive screening process that examines relevant thinking abilities. If screening indicates concerns or does not adequately characterize them, neuropsychological testing can provide a more detailed profile of affected domains. The assessment should account for communication and motor limitations, allowing clinicians to interpret results appropriately and guide individualized rehabilitation and safety planning.
Cognitive findings help the care team tailor rehabilitation to the functions that are most affected, such as attention, memory, language, executive control, or visuospatial processing. They also inform safety planning and caregiver education, so support can match the patient’s difficulties. This individualized approach connects assessment results with practical assistance rather than treating cognition as separate from recovery.
Persistent cognitive difficulties can affect how a patient manages daily activities and participates in recovery, making caregiver education an important part of coordinated care. Secondary stroke prevention is also relevant because vascular risk factors may influence cognitive outcomes and future health. Together, education, safety planning, and prevention strategies support ongoing management beyond the initial clinical assessment.
Ongoing research is seeking better biomarkers and more targeted interventions for post-stroke cognitive impairment. Improved biomarkers could help characterize or monitor cognitive effects more precisely, while targeted interventions may allow treatment and rehabilitation to align more closely with individual deficits. These advances could strengthen clinical decision-making, although current care still relies on history, screening, and selected neuropsychological testing.