Post Stroke Spasticity
Post Stroke Spasticity - Related Videos
02:59
Stereotactic Injection of Hydrogel into a Post-Stroke Mouse Model
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2025
Source: Wilson, K.L., et al. Injection of Hydrogel Biomaterial Scaffolds to The Brain After Stroke. J. Vis. Exp. (2020)This video demonstrates the stereotaxic injection of a hydrogel into the stroke core of a post-stroke mouse, highlighting its role in scaffold formation that mimics the extracellular matrix, promotes cellular infiltration, and supports angiogenesis, underscoring its neurotherapeutic potential in stroke recovery.
![Autoradiographic Measurements of [<sup>14</sup>C]-Iodoantipyrine in Rat Brain Following Central Post-Stroke Pain](/_next/image?url=https%3A%2F%2Fcloudfront.jove.com%2FCDNSource%2Fteasers%2F53947.jpg%3Fv%3D2026-09-01%252006%253A32%253A11&w=750&q=75)
07:27
Autoradiographic Measurements of [14C]-Iodoantipyrine in Rat Brain Following Central Post-Stroke Pain
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Cited by 1 •
2016
We present a protocol to measure [14C]-iodoantipyrine (IAP) uptake and assess the activation of neural substrates that are involved in central post-stroke pain (CPSP) in a rodent model.
11:34
Breathing-controlled Electrical Stimulation (BreEStim) for Management of Neuropathic Pain and Spasticity
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Cited by 9 •
2013
The purpose is to present a new method, breathing-control electrical stimulation (BreEStim) for management of neuropathic pain and spasticity.
11:06
A Human-machine-interface Integrating Low-cost Sensors with a Neuromuscular Electrical Stimulation System for Post-stroke Balance Rehabilitation
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Cited by 9 •
2016
A novel low-cost human-machine interface for interactive post-stroke balance rehabilitation system is presented in this article. The system integrates off-the-shelf low-cost sensors towards volitionally driven electrotherapy paradigm. The proof-of-concept software interface is demonstrated on healthy volunteers.
08:23
Standing Neurophysiological Assessment of Lower Extremity Muscles Post-Stroke
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Cited by 4 •
2021
This protocol describes the process for performing a neurophysiological assessment of the lower extremity muscles, tibialis anterior and soleus, in a standing position using TMS in people post-stroke. This position provides a greater probability of eliciting a post-stroke TMS response and allows for the use of reduced stimulator power during neurophysiological assessments.
FAQs
Stroke-related damage can reduce the descending inhibitory signals that normally regulate spinal reflex circuits. Without sufficient inhibition, these circuits respond excessively when muscles are stretched, contributing to increased tone and abnormal muscle activation. This neural imbalance helps explain why post-stroke movement may become less coordinated and why mobility or daily activities can require greater effort.
The resistance associated with this condition is velocity-dependent, meaning that the response to passive muscle stretching changes with the speed of movement. Clinicians therefore observe how muscles respond during controlled passive movement rather than relying only on resting appearance. This information helps characterize abnormal reflex behavior and supports decisions about rehabilitation and symptom management.
Exaggerated spinal reflex responses can increase muscle tone during movement and interfere with coordinated control. When several muscles do not respond appropriately to task demands, the person may develop inefficient or abnormal movement patterns that limit mobility. Understanding this relationship connects the observed motor problems with their neural origin and helps clinicians focus treatment on meaningful functional goals.
Assessment combines observation of movement, passive movement of affected limbs, and functional testing. These approaches show how muscle resistance appears, how abnormal tone influences available movement, and how symptoms affect practical tasks. Clinicians use the combined findings to understand the person's functional limitations and tailor an intervention plan rather than judging severity from a single observation.
Treatment is tailored to the person's movement problems, comfort, and functional needs. Clinicians may combine stretching, task-specific training, and positioning with medications or focal treatments such as botulinum toxin. This individualized approach links management to outcomes such as improved movement, greater independence, reduced discomfort, and support for long-term recovery rather than treating increased tone in isolation.
Botulinum toxin is identified as a focal treatment option within a broader management plan. Its place differs from approaches such as stretching, positioning, task-specific training, or medications, which may address rehabilitation or symptom control more broadly. Clinicians can incorporate focal treatment when tailoring care to movement limitations, with the overall aim of improving function and comfort.