Ventricular Wall
Ventricular Wall - Related Videos
07:24
Reduction in Left Ventricular Wall Stress and Improvement in Function in Failing Hearts using Algisyl-LVR
0 Views •
Cited by 34 •
2013
This article describes procedures for implanting a novel hydrogel in failing hearts and quantifying its effect on left ventricular wall stress and function. These procedures have been successfully applied in dogs and humans.
08:22
Isolation of Endocardial and Coronary Endothelial Cells from the Ventricular Free Wall of the Rat Heart
0 Views •
Cited by 13 •
2020
We present a protocol for the isolation of endocardial and coronary endothelial cells from rat hearts through sequential tissue digestion in a digestion buffer, cell collection from recurrent centrifuge cycles, and cell purification using anti-rat CD31 microbeads.
12:30
Glycan Profiling of Plant Cell Wall Polymers using Microarrays
0 Views •
Cited by 26 •
2012
A technique called Comprehensive Microarray Polymer Profiling (CoMPP) for the characterisation of plant cell wall glycans is described. This method combines the specificity of monoclonal antibodies directed to defined glycan-epitopes with a miniature microarray analytical platform allowing screening of glycan occurrence in a broad range of biological contexts.
08:05
Isolation of Embryonic Ventricular Endothelial Cells
0 Views •
Cited by 4 •
2013
Primary cell culture is a useful technique for analyzing specific populations of cells, particularly from transgenic mouse embryos at specific developmental stages. Herein, embryonic ventricles are dissected and dissociated, and antibody-conjugated beads recognize and separate out the endothelial cells for further analysis.
09:22
Chronic Thromboembolic Pulmonary Hypertension and Assessment of Right Ventricular Function in the Piglet
0 Views •
Cited by 22 •
2015
Chronic Thromboembolic Pulmonary Hypertension (CTEPH) and Right Ventricular (RV) dysfunction were induced in piglets by progressive obstruction of the pulmonary arteries. Consequences were remarkably similar to those observed in CTEPH patients. This animal model would be a very useful tool for pathophysiology and therapeutic experiments on CTEPH and RV failure.
FAQs
Electrical excitation triggers contraction of the organized cardiac muscle fibers within the myocardium. Their coordinated shortening develops pressure inside the ventricle, allowing blood to be ejected into the pulmonary artery or aorta. This relationship between electrical activation, muscle contraction, and pressure explains how ventricular wall function directly supports forward circulation.
Wall thickness provides information about ventricular workload and pumping function. Imaging may show hypertrophy, meaning increased wall thickness, or thinning, which can indicate structural change or damage. Clinicians interpret these findings alongside wall motion to assess cardiovascular disease, estimate risk, and support decisions about further evaluation or treatment.
Regional motion abnormalities identify areas of the ventricular wall that do not contract normally. Because coordinated myocardial contraction contributes to pressure generation and blood ejection, localized movement changes can signal impaired ventricular performance. These findings are especially relevant when evaluating damage after myocardial infarction and when assessing the heart’s overall pumping function.
Echocardiography and cardiac magnetic resonance provide imaging-based ways to evaluate ventricular wall structure and motion. They can reveal changes such as hypertrophy, thinning, regional motion abnormalities, or damage after myocardial infarction. The resulting information supports diagnosis, cardiovascular risk assessment, and treatment planning rather than relying only on symptoms or pressure-related assessments.
Clinical imaging focuses on measurable structural and functional features, including wall thickness and movement. It can identify whether the wall appears hypertrophied, thinned, or regionally abnormal in motion. These observations help clinicians judge ventricular workload and pumping function, recognize possible damage after myocardial infarction, and organize diagnostic or treatment decisions.
Assessment becomes particularly useful when clinicians need to evaluate cardiovascular disease, ventricular workload, or pumping performance. Imaging findings can also clarify the extent of damage after myocardial infarction and help estimate risk. By combining structural observations with wall-motion information, clinicians gain evidence that supports diagnosis and treatment planning.