Ghrelin Leptin
Ghrelin Leptin - Related Videos
05:45
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Cited by 8 •
2019
Our study focuses on the effects of leptin signaling in carotid body (CB) on the hypoxic ventilatory response (HVR). We performed 'loss of function' experiments measuring the effect of leptin on HVR after CB denervation and 'gain of function' experiments measuring HVR after overexpression of the leptin receptor in CB.
11:36
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The RAPID blood processing method can be used in humans and yields higher peptide levels as well as allows for assessment of the correct molecular form. Therefore, this method will be a valuable tool in peptide research.
11:16
Progressive-ratio Responding for Palatable High-fat and High-sugar Food in Mice
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Cited by 70 •
2012
The present report details the protocol employed to measure the rewarding effects of high-fat food in mice using a progressive ratio operant conditioning task.
10:43
Flow Cytometry Purification of Mouse Meiotic Cells
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10:55
Study of the DNA Damage Checkpoint using Xenopus Egg Extracts
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Cited by 35 •
2012
Xenopus egg extract is a useful model system to investigate the DNA damage checkpoint. This protocol is for the preparation of Xenopus egg extracts and DNA damage checkpoint inducing reagents. These techniques are adaptable to a variety of DNA damaging approaches in the study of the DNA damage checkpoint signaling.
FAQs
Researchers examine Ghrelin and Leptin together because their signals represent different metabolic states and may jointly shape eating behavior. A key psychological question is how these hormonal influences intersect with reward and motivation: appetite may reflect not only an immediate need for energy, but also the way internal metabolic information affects the value assigned to food. Stress is another relevant context for this interaction.
The timing of each signal is central to interpretation. Ghrelin typically increases during fasting, making metabolic state important when studying hunger-related behavior. Leptin provides information about stored energy, so its influence must be considered in relation to energy balance rather than treated as a simple momentary appetite signal. Together, these distinctions help connect physiological conditions with psychological responses to food.
Ghrelin and leptin are studied in relation to hypothalamic and related brain pathways that connect physiological signals with appetite regulation. This makes them useful for investigating how bodily information may influence motivation and feeding decisions. In psychology, the important issue is not simply whether a person feels hungry, but how endocrine signals may contribute to the transition from metabolic state to behavior.
Research on Ghrelin and Leptin can clarify how biological signals participate in hunger, satiety, reward, motivation, and energy balance. These findings provide a framework for examining why eating behavior varies with internal metabolic conditions, while also connecting physiological mechanisms to psychological processes. This approach avoids treating feeding behavior as purely cognitive or environmental and highlights its biological context.
Their roles in appetite regulation and energy balance provide biological context for studying obesity-related eating patterns. Research can examine how hunger and satiety signals relate to food intake and motivation without reducing obesity to a single cause. This perspective also supports investigation of interventions that target appetite regulation, linking metabolic biology with psychological research on eating behavior.
Ghrelin and leptin provide a way to investigate whether biological influences on hunger and satiety interact with psychological features of eating behavior. Because these hormones connect metabolic state with brain pathways involved in appetite, they can contribute to a broader framework for examining eating disorders. Such research considers biological and psychological influences together rather than viewing these conditions through only one lens.