Caroline A. Genco Department of Medicine, Section of Infectious Disease Boston University School of Medicine Biography Publications Institution JoVE Articles Caroline A. Genco has not added a biography. If you are Caroline A. Genco and would like to personalize this page please email our Author Liaison for assistance. Publications Porphyromonas Gingivalis Accelerates Inflammatory Atherosclerosis in the Innominate Artery of ApoE Deficient Mice Atherosclerosis. Mar, 2011 | Pubmed ID: 21251656 Pathogen-mediated Inflammatory Atherosclerosis is Mediated in Part Via Toll-like Receptor 2-induced Inflammatory Responses Journal of Innate Immunity. 2010 | Pubmed ID: 20505314 Stimulation of Toll-like Receptor 2 in Human Platelets Induces a Thromboinflammatory Response Through Activation of Phosphoinositide 3-kinase Circulation Research. Feb, 2009 | Pubmed ID: 19106411 Expression of the Gonococcal Global Regulatory Protein Fur and Genes Encompassing the Fur and Iron Regulon During in Vitro and in Vivo Infection in Women Journal of Bacteriology. May, 2008 | Pubmed ID: 18310343 Bacterial Fimbriae Stimulate Proinflammatory Activation in the Endothelium Through Distinct TLRs Journal of Immunology (Baltimore, Md. : 1950). Feb, 2008 | Pubmed ID: 18250425 Cis- and Trans-acting Elements Involved in Regulation of NorB (norZ), the Gene Encoding Nitric Oxide Reductase in Neisseria Gonorrhoeae Microbiology (Reading, England). Jan, 2008 | Pubmed ID: 18174141 Engagement of Specific Innate Immune Signaling Pathways During Porphyromonas Gingivalis Induced Chronic Inflammation and Atherosclerosis Frontiers in Bioscience : a Journal and Virtual Library. 2008 | Pubmed ID: 17981690 Toll-like Receptor 2 Plays a Critical Role in the Progression of Atherosclerosis That is Independent of Dietary Lipids Atherosclerosis. Jan, 2008 | Pubmed ID: 17466307 Porphyromonas Gingivalis Mediated Periodontal Disease and Atherosclerosis: Disparate Diseases with Commonalities in Pathogenesis Through TLRs Current Pharmaceutical Design. 2007 | Pubmed ID: 18220804 A Novel Fur- and Iron-regulated Small RNA, NrrF, is Required for Indirect Fur-mediated Regulation of the SdhA and SdhC Genes in Neisseria Meningitidis Journal of Bacteriology. May, 2007 | Pubmed ID: 17351036 Expression of the Iron-activated NspA and SecY Genes in Neisseria Meningitidis Group B by Fur-dependent and -independent Mechanisms Journal of Bacteriology. Jan, 2007 | Pubmed ID: 17085550 Hemin-dependent Modulation of the Lipid A Structure of Porphyromonas Gingivalis Lipopolysaccharide Infection and Immunity. Aug, 2006 | Pubmed ID: 16861633 Sensitization of Human Aortic Endothelial Cells to Lipopolysaccharide Via Regulation of Toll-like Receptor 4 by Bacterial Fimbria-dependent Invasion Infection and Immunity. Dec, 2005 | Pubmed ID: 16299299 Porphyromonas Gingivalis Fimbria-dependent Activation of Inflammatory Genes in Human Aortic Endothelial Cells Infection and Immunity. Sep, 2005 | Pubmed ID: 16113252 The Gonococcal Fur-regulated TbpA and TbpB Genes Are Expressed During Natural Mucosal Gonococcal Infection Infection and Immunity. Jul, 2005 | Pubmed ID: 15972520 The C-terminal Domains of the Gingipain K Polyprotein Are Necessary for Assembly of the Active Enzyme and Expression of Associated Activities Molecular Microbiology. Dec, 2004 | Pubmed ID: 15554977 Lysine-specific Gingipain K and Heme/hemoglobin Receptor HmuR Are Involved in Heme Utilization in Porphyromonas Gingivalis Acta Biochimica Polonica. 2004 | Pubmed ID: 15094847 Mice Lacking Inducible Nitric Oxide Synthase Demonstrate Impaired Killing of Porphyromonas Gingivalis Infection and Immunity. Sep, 2003 | Pubmed ID: 12933833 Immunization with Porphyromonas Gingivalis Capsular Polysaccharide Prevents P. Gingivalis-elicited Oral Bone Loss in a Murine Model Infection and Immunity. Apr, 2003 | Pubmed ID: 12654858 Мышь Модель для патогенных микроорганизмов, вызванной хроническим воспалением на локальном и системном сайтов George Papadopoulos*1, Carolyn D. Kramer*1, Connie S. Slocum1, Ellen O. Weinberg1, Ning Hua2, Cynthia V. Gudino1, James A. Hamilton1, Caroline A. Genco1 1Department of Medicine, Section of Infectious Disease, Boston University School of Medicine, 2Department of Biophysics, Boston University School of Medicine JoVE 51556 Immunology and Infection Биомолекулярных Обнаружение использованием интерферометрических датчиков изображения отражения (IRIS) Carlos A. Lopez1, George G. Daaboul2, Sunmin Ahn2, Alexander P. Reddington1, Margo R. Monroe2, Xirui Zhang2, Rostem J. Irani3, Chunxiao Yu4,5, Caroline A. Genco4,5, Marina Cretich6, Marcella Chiari6, Bennett B. Goldberg1, John H. Connor5, M. Selim Ünlü1,2 1Department of Electrical and Computer Engineering, Boston University, 2Department of Biomedical Engineering, Boston University, 3Center for Advanced Genomics Technology, Boston University, 4Department of Medicine, Section of Infectious Diseases, Boston University School of Medicine, 5Department of Microbiology, Boston University School of Medicine, 6CNR (National Research Council), Istituto di Chimica del Riconoscimento Molecolare JoVE 2694 Bioengineering
Мышь Модель для патогенных микроорганизмов, вызванной хроническим воспалением на локальном и системном сайтов George Papadopoulos*1, Carolyn D. Kramer*1, Connie S. Slocum1, Ellen O. Weinberg1, Ning Hua2, Cynthia V. Gudino1, James A. Hamilton1, Caroline A. Genco1 1Department of Medicine, Section of Infectious Disease, Boston University School of Medicine, 2Department of Biophysics, Boston University School of Medicine JoVE 51556 Immunology and Infection
Биомолекулярных Обнаружение использованием интерферометрических датчиков изображения отражения (IRIS) Carlos A. Lopez1, George G. Daaboul2, Sunmin Ahn2, Alexander P. Reddington1, Margo R. Monroe2, Xirui Zhang2, Rostem J. Irani3, Chunxiao Yu4,5, Caroline A. Genco4,5, Marina Cretich6, Marcella Chiari6, Bennett B. Goldberg1, John H. Connor5, M. Selim Ünlü1,2 1Department of Electrical and Computer Engineering, Boston University, 2Department of Biomedical Engineering, Boston University, 3Center for Advanced Genomics Technology, Boston University, 4Department of Medicine, Section of Infectious Diseases, Boston University School of Medicine, 5Department of Microbiology, Boston University School of Medicine, 6CNR (National Research Council), Istituto di Chimica del Riconoscimento Molecolare JoVE 2694 Bioengineering
