Julie A. Sterling Vanderbilt Center for Bone Biology Vanderbilt University Biography Publications Institution JoVE Articles Julie A. Sterling has not added a biography. If you are Julie A. Sterling and would like to personalize this page please email our Author Liaison for assistance. Publications Stimulation of Host Bone Marrow Stromal Cells by Sympathetic Nerves Promotes Breast Cancer Bone Metastasis in Mice PLoS Biology. Jul, 2012 | Pubmed ID: 22815651 Loss of TGF-β Responsiveness in Prostate Stromal Cells Alters Chemokine Levels and Facilitates the Development of Mixed Osteoblastic/osteolytic Bone Lesions Molecular Cancer Research : MCR. Apr, 2012 | Pubmed ID: 22290877 6-Thioguanine Inhibition of Parathyroid Hormone-related Protein Expression is Mediated by GLI2 Anticancer Research. Sep, 2011 | Pubmed ID: 21868511 Contribution of Bone Tissue Modulus to Breast Cancer Metastasis to Bone Cancer Microenvironment : Official Journal of the International Cancer Microenvironment Society. Dec, 2011 | Pubmed ID: 21789687 Bone Structural Components Regulating Sites of Tumor Metastasis Current Osteoporosis Reports. Jun, 2011 | Pubmed ID: 21424744 TGF-beta Promotion of Gli2-induced Expression of Parathyroid Hormone-related Protein, an Important Osteolytic Factor in Bone Metastasis, is Independent of Canonical Hedgehog Signaling Cancer Research. Feb, 2011 | Pubmed ID: 21189326 Longitudinal Live Animal Micro-CT Allows for Quantitative Analysis of Tumor-induced Bone Destruction Bone. Jan, 2011 | Pubmed ID: 20685406 Advances in the Biology of Bone Metastasis: How the Skeleton Affects Tumor Behavior Bone. Jan, 2011 | Pubmed ID: 20643235 Matrix Rigidity Induces Osteolytic Gene Expression of Metastatic Breast Cancer Cells PloS One. 2010 | Pubmed ID: 21085597 The Hedgehog Signaling Molecule Gli2 Induces Parathyroid Hormone-related Peptide Expression and Osteolysis in Metastatic Human Breast Cancer Cells Cancer Research. Aug, 2006 | Pubmed ID: 16885353 PARP Regulates TGF-beta Receptor Type II Expression in Estrogen Receptor-positive Breast Cancer Cell Lines Anticancer Research. May-Jun, 2006 | Pubmed ID: 16827122 Kemik Metastaz Modelleri J. Preston Campbell1,2, Alyssa R. Merkel2,3,4, S. Kathryn Masood-Campbell2,4, Florent Elefteriou1,2,4,5, Julie A. Sterling2,3,4,5 1Department of Pharmacology, Vanderbilt University, 2Vanderbilt Center for Bone Biology, Vanderbilt University, 3Department of Veterans Affairs, Tennessee Valley Healthcare System (VISN 9), 4Department of Medicine, Division of Clinical Pharmacology, Vanderbilt University, 5Department of Cancer Biology, Vanderbilt University JoVE 4260 Medicine
Kemik Metastaz Modelleri J. Preston Campbell1,2, Alyssa R. Merkel2,3,4, S. Kathryn Masood-Campbell2,4, Florent Elefteriou1,2,4,5, Julie A. Sterling2,3,4,5 1Department of Pharmacology, Vanderbilt University, 2Vanderbilt Center for Bone Biology, Vanderbilt University, 3Department of Veterans Affairs, Tennessee Valley Healthcare System (VISN 9), 4Department of Medicine, Division of Clinical Pharmacology, Vanderbilt University, 5Department of Cancer Biology, Vanderbilt University JoVE 4260 Medicine