Moshmi Bhattacharya Department of Physiology and Pharmacology, Schulich School of Medicine and Dentistry University of Western Ontario Biography Publications Institution JoVE Articles Moshmi Bhattacharya has not added a biography. If you are Moshmi Bhattacharya and would like to personalize this page please email our Author Liaison for assistance. Publications Implantation Failure in Female Kiss1(-/-) Mice is Independent of Their Hypogonadic State and Can Be Partially Rescued by Leukemia Inhibitory Factor Endocrinology. May, 2014 | Pubmed ID: 24877624 Nodal Signals Via β-arrestins and RalGTPases to Regulate Trophoblast Invasion Cellular Signalling. May, 2014 | Pubmed ID: 24863882 Kisspeptin/KISS1R Signaling Potentiates Extravillous Trophoblast Adhesion to Type-I Collagen in a PKC- and ERK1/2-dependent Manner Molecular Reproduction and Development. Jan, 2014 | Pubmed ID: 24273038 Kisspeptin/KISS1R System in Breast Cancer Journal of Cancer. 2013 | Pubmed ID: 24155777 KISS1R Induces Invasiveness of Estrogen Receptor-Negative Human Mammary Epithelial and Breast Cancer Cells Endocrinology. Mar, 2013 | Pubmed ID: 23525242 β-Arrestin2 Regulates Lysophosphatidic Acid-induced Human Breast Tumor Cell Migration and Invasion Via Rap1 and IQGAP1 PloS One. 2013 | Pubmed ID: 23405264 βarrestin2 Interacts with TβRII to Regulate Smad-dependent and Smad-independent Signal Transduction Cellular Signalling. Jan, 2013 | Pubmed ID: 23069267 Single-cell Analyses Reveal That KISS1R-expressing Cells Undergo Sustained Kisspeptin-induced Signaling That is Dependent Upon an Influx of Extracellular Ca2+ Endocrinology. Dec, 2012 | Pubmed ID: 23070548 GPR54 (KISS1R) Transactivates EGFR to Promote Breast Cancer Cell Invasiveness PloS One. 2011 | Pubmed ID: 21738726 GPR54 Regulates ERK1/2 Activity and Hypothalamic Gene Expression in a Gα(q/11) and β-arrestin-dependent Manner PloS One. 2010 | Pubmed ID: 20886089 The Human Gonadotropin Releasing Hormone Type I Receptor is a Functional Intracellular GPCR Expressed on the Nuclear Membrane PloS One. 2010 | Pubmed ID: 20628612 Regulation of GPR54 Signaling by GRK2 and {beta}-arrestin Molecular Endocrinology (Baltimore, Md.). Dec, 2009 | Pubmed ID: 19846537 Beta-arrestin/Ral Signaling Regulates Lysophosphatidic Acid-mediated Migration and Invasion of Human Breast Tumor Cells Molecular Cancer Research : MCR. Jul, 2009 | Pubmed ID: 19609003 Gonadotropin-releasing Hormone-regulated Chemokine Expression in Human Placentation American Journal of Physiology. Cell Physiology. Jul, 2009 | Pubmed ID: 19369450 Dual Regulation of Lysophosphatidic Acid (LPA1) Receptor Signalling by Ral and GRK Cellular Signalling. Jul, 2009 | Pubmed ID: 19306925 Ral and Phospholipase D2-dependent Pathway for Constitutive Metabotropic Glutamate Receptor Endocytosis The Journal of Neuroscience : the Official Journal of the Society for Neuroscience. Oct, 2004 | Pubmed ID: 15470141 Identification of a Novel Nuclear Localization Signal Common to 69- and 82-kDa Human Choline Acetyltransferase The Journal of Biological Chemistry. May, 2003 | Pubmed ID: 12637523 Beta-arrestins Regulate a Ral-GDS Ral Effector Pathway That Mediates Cytoskeletal Reorganization Nature Cell Biology. Aug, 2002 | Pubmed ID: 12105416 एक तीन आयामी (3 डी) मॉडल का उपयोग कर स्तन कैंसर कोशिका invasiveness की मात्रा Donna Cvetković*1, Cameron Glenn-Franklin Goertzen*1, Moshmi Bhattacharya1,2,3 1Department of Physiology and Pharmacology, Schulich School of Medicine and Dentistry, University of Western Ontario, 2Department of Oncology, Schulich School of Medicine and Dentistry, University of Western Ontario, 3Lawson Health Research Institute JoVE 51341 Medicine
एक तीन आयामी (3 डी) मॉडल का उपयोग कर स्तन कैंसर कोशिका invasiveness की मात्रा Donna Cvetković*1, Cameron Glenn-Franklin Goertzen*1, Moshmi Bhattacharya1,2,3 1Department of Physiology and Pharmacology, Schulich School of Medicine and Dentistry, University of Western Ontario, 2Department of Oncology, Schulich School of Medicine and Dentistry, University of Western Ontario, 3Lawson Health Research Institute JoVE 51341 Medicine
