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Journal / Biology / 缺乏PMS2，ERCC1，Ku86，视野缺损CcOI在结肠癌进展…
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缺乏PMS2，ERCC1，Ku86，视野缺损CcOI在结肠癌进展
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缺乏PMS2，ERCC1，Ku86，视野缺损CcOI在结肠癌进展

Article DOI: 10.3791/1931-v • 28:16 min • July 28th, 2010
July 28th, 2010 •
1, 1, 1, 1, 1, 1, 2,3, 1,4, 3, 5, 5, 5, 1
1Department of Cell Biology and Anatomy, College of Medicine, University of Arizona, Tucson, 2Southern Arizona Veterans Affairs Health Care System, Tucson, AZ, 3Department of Surgery, College of Medicine, University of Arizona, Tucson, 4Biomedical Diagnostics and Research, Tucson, AZ, 5Department of Medicine, College of Medicine, University of Arizona, Tucson

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减少/缺失PMS2和/或整个隐窝ERCC1的表达是在10厘米的结肠腺癌，可能与高可变性和发展为癌症的视野缺损的基础上两侧的频繁事件。 Ku86或CcOI虚寒经常在这些领域的缺陷要少得多。

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细胞生物学，第41期，DNA修复，细胞凋亡，视野缺损，结肠癌，PMS2，ERCC1基因，细胞色素C氧化酶亚基我，Ku86，免疫组织化学，手术切除
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