Thibault Voeltzel Centre de Recherche en Cancérologie de Lyon Biography Publications Institution JoVE Articles Thibault Voeltzel has not added a biography. If you are Thibault Voeltzel and would like to personalize this page please email our Author Liaison for assistance. Publications Erratum for Brahimi-Horn Et Al., "Local Mitochondrial-Endolysosomal Microfusion Cleaves the Voltage-Dependent Anion Channel 1 To Promote Survival in Hypoxia" Molecular and Cellular Biology. Jan, 2022 | Pubmed ID: 35049340 A Minimal Standardized Human Bone Marrow Microphysiological System to Assess Resident Cell Behavior During Normal and Pathological Processes Biomaterials Science. Jan, 2022 | Pubmed ID: 34904143 Altered BMP2/4 Signaling in Stem Cells and Their Niche: Different Cancers but Similar Mechanisms, the Example of Myeloid Leukemia and Breast Cancer Frontiers in Cell and Developmental Biology. 2021 | Pubmed ID: 35047500 The Quiescent Fraction of Chronic Myeloid Leukemic Stem Cells Depends on BMPR1B, Stat3 and BMP4-niche Signals to Persist in Patients in Remission Haematologica. Jan, 2021 | Pubmed ID: 32001529 A New Signaling Cascade Linking BMP4, BMPR1A, ΔNp73 and NANOG Impacts on Stem-like Human Cell Properties and Patient Outcome Cell Death & Disease. Sep, 2018 | Pubmed ID: 30262802 The BMP Pathway: A Unique Tool to Decode the Origin and Progression of Leukemia Experimental Hematology. May, 2018 | Pubmed ID: 29477370 Immature CML Cells Implement a BMP Autocrine Loop to Escape TKI Treatment Blood. Dec, 2017 | Pubmed ID: 29138221 Local Mitochondrial-endolysosomal Microfusion Cleaves Voltage-dependent Anion Channel 1 to Promote Survival in Hypoxia Molecular and Cellular Biology. May, 2015 | Pubmed ID: 25691661 Disequilibrium of BMP2 Levels in the Breast Stem Cell Niche Launches Epithelial Transformation by Overamplifying BMPR1B Cell Response Stem Cell Reports. Feb, 2015 | Pubmed ID: 25601208 Primitive CML Cell Expansion Relies on Abnormal Levels of BMPs Provided by the Niche and on BMPRIb Overexpression Blood. Nov, 2013 | Pubmed ID: 24100446 Ein menschliches Knochenmark-3D-Modell zur Untersuchung der Dynamik und Interaktionen zwischen residenten Zellen in physiologischen oder tumoralen Kontexten Kawtar Arizkane1,2,3, Kevin Geistlich1,2,3,4, Laurine Moindrot1,2,3, Emma Risson1,2,3, Sandrine Jeanpierre1,2,3,4, Léa Barral1,2,3, Anaëlle Bobard1,2,3, Giulio Menegazzi5, Thibault Voeltzel1,2,3, Véronique Maguer-Satta1,2,3, Sylvain Lefort1,2,3 1Centre de Recherche en Cancérologie de Lyon, 2Centre de Recherche en Cancérologie de Lyon, 3Université de Lyon, 4Centre Léon Bérard, 5Department of Experimental and Clinical Biomedical Sciences Mario Serio, Università degli Studi di Firenze JoVE 64736 Biology
Ein menschliches Knochenmark-3D-Modell zur Untersuchung der Dynamik und Interaktionen zwischen residenten Zellen in physiologischen oder tumoralen Kontexten Kawtar Arizkane1,2,3, Kevin Geistlich1,2,3,4, Laurine Moindrot1,2,3, Emma Risson1,2,3, Sandrine Jeanpierre1,2,3,4, Léa Barral1,2,3, Anaëlle Bobard1,2,3, Giulio Menegazzi5, Thibault Voeltzel1,2,3, Véronique Maguer-Satta1,2,3, Sylvain Lefort1,2,3 1Centre de Recherche en Cancérologie de Lyon, 2Centre de Recherche en Cancérologie de Lyon, 3Université de Lyon, 4Centre Léon Bérard, 5Department of Experimental and Clinical Biomedical Sciences Mario Serio, Università degli Studi di Firenze JoVE 64736 Biology