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アポ E 欠損マウスにおける鉱質コルチコイド受容体の活性化を介して動脈硬化性プラークの誘導
VDJ-Seq: Deep Sequencing Analysis of Rearranged Immunoglobulin Heavy Chain Gene to Reveal Clonal Evolution Patterns of B Cell Lymphoma
Journal (Medicine)
VDJ-Seq: Deep Sequencing Analysis of Rearranged Immunoglobulin Heavy Chain Gene to Reveal Clonal Evolution Patterns of B Cell Lymphoma
A Model of Experimental Steatosis In Vitro: Hepatocyte Cell Culture in Lipid Overload-Conditioned Medium
Journal (Medicine)
A Model of Experimental Steatosis In Vitro: Hepatocyte Cell Culture in Lipid Overload-Conditioned Medium
 
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アポ E 欠損マウスにおける鉱質コルチコイド受容体の活性化を介して動脈硬化性プラークの誘導

Article DOI: 10.3791/58303-v • 07:36 min • September 26th, 2018
September 26th, 2018 •
1, 1, 1, 1, 1,2
1Laboratory of Cardiovascular Endocrinology, IRCCS San Raffaele Pisana, Rome, Italy, 2Department of Human Sciences and Promotion of the Quality of Life, San Raffaele Roma Open University, Rome, Italy

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アルドステロンの連続リリースで、動脈硬化の食事と飼育アポ-/-マウスの大動脈における動脈硬化を誘発するプロトコルについて述べる。プラークの組成の同定手法について述べる。

Tags

医学、問題 139、アテローム性動脈硬化、アルドステロン、炎症、アポ-/-、大動脈、免疫組織化学
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