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缺乏PMS2,ERCC1,Ku86,视野缺损CcOI在结肠癌进展
JoVE Journal
Biology
This content is Free Access.
JoVE Journal
Biology
Deficient Pms2, ERCC1, Ku86, CcOI in Field Defects During Progression to Colon Cancer
Please note that all translations are automatically generated.
Click here for the English version.
缺乏PMS2,ERCC1,Ku86,视野缺损CcOI在结肠癌进展
DOI:
10.3791/1931-v
•
28:15 min
•
July 28, 2010
•
Huy Nguyen
,
Cristy Loustaunau
,
Alexander Facista
,
Lois Ramsey
,
Nadia Hassounah
,
Hilary Taylor
,
Robert Krouse
3
,
Claire M. Payne
4
,
V. Liana Tsikitis
,
Steve Goldschmid
,
Bhaskar Banerjee
,
Rafael F. Perini
,
Carol Bernstein
1
Department of Cell Biology and Anatomy, College of Medicine
,
University of Arizona, Tucson
,
2
Southern Arizona Veterans Affairs Health Care System
,
Tucson, AZ
,
3
Department of Surgery, College of Medicine
,
University of Arizona, Tucson
,
4
Biomedical Diagnostics and Research
,
Tucson, AZ
,
5
Department of Medicine, College of Medicine
,
University of Arizona, Tucson
Chapters
00:05
Title
01:47
Introduction
03:04
Preparing Tissues
04:15
Immunostaining
08:16
Evaluating Tissue Sections
27:11
Conclusion
Summary
Automatic Translation
English (Original)
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français (French)
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português (Portuguese)
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Automatic Translation
减少/缺失PMS2和/或整个隐窝ERCC1的表达是在10厘米的结肠腺癌,可能与高可变性和发展为癌症的视野缺损的基础上两侧的频繁事件。 Ku86或CcOI虚寒经常在这些领域的缺陷要少得多。
Tags
Deficient Pms2
Deficient ERCC1
Deficient Ku86
Deficient CcOI
Field Defect
Colon Cancer
Carcinogenesis
Molecular Abnormalities
Colonic Adenocarcinoma
Adenoma With Advanced Neoplasia
Immunohistochemistry
DNA Repair Proteins
Apoptosis
DNA Mismatch Repair
DNA Nucleotide Excision Repair
Apoptosis Resistance
Mutability
Progression To Colon Cancer
DNA Non-homologous End Joining
Cytochrome C Oxidase Subunit
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